USDA Associate Professor, NCSU Department of Pathology
Type: Associate Faculty
PhD, John Innes Centre, UK
Postdoctoral, National Institutes of Health and Cornell University
Office: 2572 Thomas Hall
Website: Visit our Lab Home Page
We work on the genetic and molecular basis of plant disease resistance and the defense response. In particular we are interested in the genetic basis of quantitative or partial resistance in maize and in the genetic architecture controlling variation in the maize defense response. We are also interested in genes conferring multiple disease resistance.
Wisser R, Kolkman J, Patzoldt M, Holland J, Yu J, Krakowsky M, Nelson R, and Balint-Kurti P. (2011). Multivariate analysis of resistances to three diseases in maize suggests a pleiotropic genetic basis and implicates a glutathione S-transferase as a multiple disease resistance gene. Proceedings of the National Academy of Sciences. 108:7339–7344.
Kump K, Bradbury P, Buckler E, Belcher A, Oropeza-Rosas M, Wisser R, Zwonitzer J, Kresovich S, McMullen M, Ware D, Balint-Kurti P, and Holland J. (2011). Genome-wide association study of quantitative resistance to southern leaf blight in the maize nested association mapping population. Nature Genetics. 43:163–168.
Kump K, Holland J, Jung M, Wolters P, and Balint-Kurti P. (2010). Joint analysis of near isogenic and recombinant inbred line populations yields precise positional estimates for QTL. Plant Genome. 3:142–153.
Chintamanani S, Hulbert SH, Johal GS, and Balint-Kurti P. (2010). Identification of a Maize Locus that Modulates the Hypersensitive Defense Response, Using Mutant-Assisted Gene Identification and Characterization (MAGIC). Genetics. 184:813–825.
Balint-Kurti PJ, Zwonitzer JC, Wisser RJ, Carson ML, Oropeza-Rosas M, Holland JB, and Szalma SJ. (2007). Precise mapping of quantitative trait loci for resistance to southern leaf blight, caused by Cochliobolus heterostrophus race O, and flowering time using advanced intercross maize lines. Genetics. 176:645–657.